Summary

of the entire

“Excessive Calcium Causes Osteoporosis”

website

 

 

 

 

 

 

National institutes and doctors advice you to consume high calcium foods and/or to take calcium supplements.

Their advice is based on the hypothesis that it may prevent osteoporosis. They reason that this theory is supported by studies that show that consuming extra calcium increases bone mass and bone strength. 

 

However, these studies only cover the short-term effects of the extra calcium, not the life time effects, while osteoporosis is an aging disease; a disease that only shows its face after decades of living a certain lifestyle.

Concluding, from these studies, that extra calcium may prevent osteoporosis, is like concluding that bodybuilding may prevent muscle diseases. Yes, a short term effect of bodybuilding is that your muscles grow bigger and stronger, but that does not mean that these current activities are still beneficial to your body when you are 70 years old.

 

There are no studies about the lifetime effects of diet on bone health, but there ARE international statistics; about the food people in a certain country eat, from the crib to the grave, and about how often old people in that country break their hip. By comparing these data, you learn about the lifetime effects of diet on bone health.

And guess what? 

International statistics show that in all countries where the most calcium is consumed, the hip fracture rates are the highest (these rates are independent of the % of old people in a country). 

So, the LIFETIME effects of consuming much calcium are detrimental.

 

To overcome this contradiction, they simply changed the definition of osteoporosis, from "irreversible porosity of the bones" to: "low bone mineral mass". Bones however, can very well have a low mass while being perfectly healthy and strong.

Under this new definition, bone health is no longer important, nor fracture rates, but bone mass only. Under this new definition, there is LESS osteoporosis in countries where old people averagely break their hip MOST OFTEN.

Under this new definition, people with healthy, strong bones, but with a low bone mass, have osteoporosis.

Every construction engineer can tell you that the strength of a construction is certainly NOT solely dependent on the thickness of the material used.

 

So, what is it that they are trying to hide so desperately?

That they have been giving bad advice for decades?

With totally adverse effects?

 

International statistics indicate that the lifetime effects of extra calcium (through dairy consumption) are detrimental.

 

How can extra calcium can have detrimental lifetime effects on our bone cells?

Maybe just like extra sunlight exposure has detrimental long term effects on your skin?

 

Even that a tan makes you look vibrant and healthy as you are young, your own eyes can see that the lifetime effects of extra sun exposure to a white skin are detrimental (think of those 'orange-colored' and wrinkled people that look way too old for their age)  

 

What is the corresponding factor?

 

The aging process. 

Both skin cells and bone cells age. And the number of times that these cells can be renewed, is limited in both. This means that the faster they age, the sooner their renewal capacity will eventually be exhausted.

Skin cells age due to the chemicals and sunlight exposure that they have to endure, which dehydrates the outer cells, stimulating the replacement of the cells that have died.

Bone cells age due to the calcium that they have to process, and due to the strain they have to endure. the more calcium they have to process, or strain to endure, to more cells die, and the renewal of these cells is accelerated.  

 

How can you prove this?

 

Through a simple (low cost) experiment

 

 

 

'Circumstantial Evidence' for the theory mentioned above:

 

A : In all countries where the most calcium is consumed, the hip fracture incidence is highest too

B : The more osteoblast activity (and proliferation and apoptosis) is increased, the higher the bone mineral density (bone 'mass'). The hip fracture incidence is the highest in countries where the average bone mass is highest too.

C : Adequate natural lifetime estrogen production has been shown to be protective against osteoporosis, while estrogen INHIBITS the uptake of calcium into the bones.

D : Prolonged intake of corticosteroids has been shown to increase osteoporosis risk. Corticosteroids have been shown to kill osteoblasts, thus stimulating the renewal of these killed cells, accelerating the aging process of osteoblasts. 

E : Bone cells don't just age due to the processing of calcium, but also due to physical strain. Professional athletes have been shown to be susceptible to 'stress fractures', due to intense physical training. The more intense the exercises, the more bone cells are destroyed, stimulating the renewal of bone cells, accelerating the aging process of osteoblasts. 

F : Hyperparathyroidism has been shown to cause osteoporosis eventually. Parathyroid hormone (PTH) has been shown to INCREASE osteoblast activity (and apoptosis; accelerating the aging process) Vitamin D decreases too high PTH levels.

G : Fluoride intake is moderately related to hip fracture incidence. Fluoride can increase osteoblast cell proliferation and bone mineral density

H : Protein intake is moderately related to hip fracture incidence. Protein stimulates osteoblast cell proliferation and can increase bone mineral density

I : Overweight seems to be protective against osteoporosis. Because the fatter you become, the more leptin is secreted to inhibit your appetite. This hormone also inhibits bone formation by osteoblasts (slowing down the aging process) 

Though the overweight itself causes a higher strain on the bones, this is compensated by the fact that overweight people sport less. 

 

J : All the other theories about osteoporosis appear to be inconsistent.

 

 

 

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